Fig. 8. Complementation between different diß point mutations. Expression of diß in the wing caused a dominant negative wing-blister phenotype (A) that was lost in the mutant diß constructs (B), e.g. P841A. Dominant negative activity was restored by coexpression of two different diß mutants. The diß mutants P829A and P841A produced a weak wrinkled-wing phenotype (C), whereas N828A and P841A restored a wing-blister phenotype (D).