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Fig. 7. In vivo distribution (A) and dynamics (B) of HMGA1a-GFP proteins after inhibition of p34 by roscovitine (a), after inhibition of protein kinase C (PKC) by a specific cell permeable inhibitory peptide (b) and after inhibition of histone deacetylases by Trichostatin A [TSA, shown in (c)]. Note the altered distribution of HMGA1a-GFP after the different drug treatments. Bar represents 10 µm. (B) Recovery kinetics of HMGA1a-GFP after bleaching of drug treated cells. Treatment of cells with Roscovitin, PKC inhibitor or TSA increased the HMGA1a-GFP mobility compared with untreated cells.





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