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Fig. 5. Mutant cells defective in pathways affecting non-homologous end-joining, the DNA-damage checkpoint, replication-fork stability and the MRN complex appear to produce less ROS than mutant cells defective in replication proteins. The indicated strains were incubated at 25°C for 80 minutes in the presence of DCDHFDA. Other conditions were as described in Figs 1 and 4.





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