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Figure 7


Fig. 7. (A) In the wild-type state, signaling through DAF-2, AGE-1 and IFTA-2 leads to phosphorylated DAF-16, which remains cytoplasmic and results in wild-type lifespan and wild-type dauer formation. (B) In ifta-2 mutants, disruption of the pathway between DAF-2 and DAF-16 leads to an accumulation of DAF-16 in the nucleus, extended lifespan and altered dauer formation.





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