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Figure 8


Fig. 8. Effect of various inhibitors on high-K+- and agonist-induced contractions in fresh rat aorta tissues. (A) Ca2+-free, EGTA-containing solution inhibited the development of contractions induced by high K+. (B) The first force trace shows a control contraction, transient and practically irreversible, induced by 1 µM ATII after a contraction induced by high K+. The second trace (from another tissue strip) shows that the development of transient ATII-induced contraction is prevented by in presence of 30 µM 2-APB. (C) ET-1 (1 µM) produced a large sustained contraction, which was relaxed to the base line by 3 µM GF-109203X, 30 µM 2-APB and 10 µM Y-27632. (D) PDBu (1 µM) generated a large contraction, which was reduced by 3 µM GF-109203X. All figures are representative of 3-5 similar experiments.





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