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Fig. 3. Ectopic spreading of H3K9me2 in dAda2a, dGcn5 and JIL-1 mutants. (A) Polytene chromosome squashes of dAda2a^d189-, dGcn5^E333st- and JIL-1^z2-null homozygous third-instar larvae stained with H3K9me2-specific antibody and DAPI (blue). (B) Polytene nuclei of dAda2a^d189-, dGcn5^E333st- and JIL-1^z2-null homozygous larvae stained with H3K9me2-specific antibody (red) and DAPI (blue). (C) Su(var)3-9 heterozygocy increases the viability of dAda2a- or dGcn5-null animals. The percentages of animals perishing at the indicated developmental stages are shown. The number of animals in each category was over 350. (D) Su(var)3-9 heterozygous background improves the structure of male X chromosomes of dAda2a-null mutants.

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