|
|
|
||||
| Home Help Feedback Subscriptions Archive Search Table of Contents | |||||
This article has been cited by other articles:
![]() |
O. M. Hernandez, M. Jones, G. Guzman, and D. Szczesna-Cordary Myosin essential light chain in health and disease Am J Physiol Heart Circ Physiol, April 1, 2007; 292(4): H1643 - H1654. [Abstract] [Full Text] [PDF] |
||||
![]() |
P. Young, E. Ehler, and M. Gautel Obscurin, a giant sarcomeric Rho guanine nucleotide exchange factor protein involved in sarcomere assembly J. Cell Biol., July 9, 2001; 154(1): 123 - 136. [Abstract] [Full Text] [PDF] |
||||
![]() |
P. F.M. van der Ven, S. Wiesner, P. Salmikangas, D. Auerbach, M. Himmel, S. Kempa, K. Haye{beta}, D. Pacholsky, A. Taivainen, R. Schroder, et al. Indications for a Novel Muscular Dystrophy Pathway: {gamma}-Filamin, the Muscle-specific Filamin Isoform, Interacts with Myotilin J. Cell Biol., October 9, 2000; 151(2): 235 - 248. [Abstract] [Full Text] [PDF] |
||||
![]() |
D. Auerbach, S. Bantle, S. Keller, V. Hinderling, M. Leu, E. Ehler, and J.-C. Perriard Different Domains of the M-Band Protein Myomesin Are Involved in Myosin Binding and M-Band Targeting Mol. Biol. Cell, May 1, 1999; 10(5): 1297 - 1308. [Abstract] [Full Text] |
||||
![]() |
M. Suomalainen, M. Y. Nakano, S. Keller, K. Boucke, R. P. Stidwill, and U. F. Greber Microtubule-dependent Plus- and Minus End-directed Motilities Are Competing Processes for Nuclear Targeting of Adenovirus J. Cell Biol., February 22, 1999; 144(4): 657 - 672. [Abstract] [Full Text] [PDF] |
||||
![]() |
D. Helfman, C Berthier, J Grossman, M Leu, E Ehler, E Perriard, and J. Perriard Nonmuscle tropomyosin-4 requires coexpression with other low molecular weight isoforms for binding to thin filaments in cardiomyocytes J. Cell Sci., January 2, 1999; 112(3): 371 - 380. [Abstract] [PDF] |
||||
![]() |
K. D. Becker, K. R. Gottshall, R. Hickey, J.-C. Perriard, and K. R. Chien Point Mutations in Human beta Cardiac Myosin Heavy Chain Have Differential Effects on Sarcomeric Structure and Assembly: An ATP Binding Site Change Disrupts Both Thick and Thin Filaments, Whereas Hypertrophic Cardiomyopathy Mutations Display Normal Assembly J. Cell Biol., April 7, 1997; 137(1): 131 - 140. [Abstract] [Full Text] [PDF] |
||||
| |||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||||